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Hyperphosphorylation of the tau protein (tau inclusions, pTau) can result in the self-assembly of tangles of paired helical filaments and straight filaments, which are involved in the pathogenesis of Alzheimer's disease, frontotemporal dementia, and other
Toxic form of tau protein foils memory formation in Alzheimer’s. Study in mice provides a mechanism for how the notorious protein stops neurons from strengthening their connections – and how we form memories